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<title>Phlebology current issue</title>
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<description>Phlebology RSS feed -- current issue</description>
<prism:coverDisplayDate>October 2009</prism:coverDisplayDate>
<prism:publicationName>Phlebology</prism:publicationName>
<prism:issn>0268-3555</prism:issn>
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<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/191?rss=1">
<title><![CDATA[The Australasian College of Phlebology and its role]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/191?rss=1</link>
<description><![CDATA[]]></description>
<dc:creator><![CDATA[Thibault, P K]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.09e004</dc:identifier>
<dc:title><![CDATA[The Australasian College of Phlebology and its role]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>192</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>191</prism:startingPage>
<prism:section>Editorial</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/193?rss=1">
<title><![CDATA[Arterio-venous malformation: how much do we know?]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/193?rss=1</link>
<description><![CDATA[
<p>Arterio-venous malformation (AVM) is a congenital vascular malformation that is neither a venous malformation nor a haemangioma. An AVM is a potentially life-threatening and limb-threatening lesion, especially the &lsquo;fistulous&rsquo; truncular form due to its unique embryological and haemodynamic characteristics. AVM treatment requires an early aggressive approach, one that is careful and based on a thorough assessment of the risks and benefits associated with the treatment plan.</p>
<p>A successful treatment strategy requires an accurate assessment of the AVM taking into account the extent, severity and progression of the lesion. This is critical in order to minimize the morbidity associated with the currently available therapies. A multidisiciplinary approach that integrates endovascular and surgical therapy can substantially improve the treatment results seen in patients with AVMs. Preliminary treatment of a fistulous AVM with coil embolization is essential in order to minimize associated morbidity and to alter the lesion haemodynamics from a high-flow lesion to a low-flow lesion that is more amenable to subsequent, definitive management with ethanol or NBCA glue embolo/sclerotherapy.</p>
]]></description>
<dc:creator><![CDATA[Lee, B B, Lardeo, J, Neville, R]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.009032</dc:identifier>
<dc:title><![CDATA[Arterio-venous malformation: how much do we know?]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>200</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>193</prism:startingPage>
<prism:section>Venous Disease A-Z series: no. 12</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/201?rss=1">
<title><![CDATA[Popliteal vein compression syndrome: obesity, venous disease and the popliteal connection]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/201?rss=1</link>
<description><![CDATA[
<sec><st>Objectives</st>
<p>Obesity and venous disease are commonly encountered together. The aetio- logical relationship, however, has not been clear. Popliteal venous compression (PVC) has been encountered both on ultrasound and venographically. In this study, patients with symptoms and/or signs of chronic venous hypertension with PVC were investigated and the relationship to obesity was defined.</p>
</sec>
<sec><st>Methods</st>
<p>A total of 89 patients were included in the study, of which 49 limbs were classified as having PVC defined as a greater than 90% reduction in the maximum internal diameter (ID) of the popliteal vein (POPV) with knee locking. Forty consecutive limbs with venous disease with no evidence of PVC were used as controls. The body mass index (BMI) of each group was calculated and the clinical symptoms and signs were documented. After the failure of conservative treatment, 30 of the 49 underwent open popliteal decompression.</p>
</sec>
<sec><st>Results</st>
<p>Patients with PVC were found to have a BMI of 34.6 &plusmn; 6.2 compared with 25.3 &plusmn; 3.0 of the controls. The POPV ID in the PVC group before and after knee locking changed from 11.7 &plusmn; 5.0 to 1.0 &plusmn; 2.1 mm, respectively. Postoperatively, the POPV ID before and after knee locking changed from 10.2 &plusmn; 2.2 to 9.0 &plusmn; 1.5 mm, respectively. At 16.2 &plusmn; 12.1 months follow-up, all the major clinical parameters improved at a statistically significant level.</p>
</sec>
<sec><st>Conclusions</st>
<p>There appears to be a relationship between obesity, chronic venous disease and PVC. POPV compression syndrome may clarify the previously unexplained venous presentations. Surgical decompression provides good results in patients unresponsive to conservative treatment.</p>
</sec>
]]></description>
<dc:creator><![CDATA[Lane, R J, Cuzzilla, M L, Harris, R A, Phillips, M N]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2008.008039</dc:identifier>
<dc:title><![CDATA[Popliteal vein compression syndrome: obesity, venous disease and the popliteal connection]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>207</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>201</prism:startingPage>
<prism:section>Original articles</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/208?rss=1">
<title><![CDATA[Treatment of incompetent perforating veins using the radiofrequency ablation stylet: a pilot study]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/208?rss=1</link>
<description><![CDATA[
<sec><st>Background</st>
<p>Although the role of incompetent perforating veins (IPV) in chronic venous insufficiency remains controversial, they are often treated by surgical or by minimal invasive techniques.</p>
</sec>
<sec><st>Objectives</st>
<p>To describe the procedure of radiofrequency ablation (RFA) of IPV and to evaluate its short-term effectiveness and safety.</p>
</sec>
<sec><st>Methods</st>
<p>In a clinical pilot study, 14 IPV in 12 patients were treated with a radiofrequency stylet. After three months, ultrasound (US) examination was used to assess anatomical success rate and exclude deep venous thrombosis. Also, self-reported side-effects were investigated.</p>
</sec>
<sec><st>Results</st>
<p>Of the 14 treated IPV, nine (64%) were obliterated on US examination and the others showed remaining reflux. Two patients reported localized paresthesia, but no deep venous thrombosis was recorded.</p>
</sec>
<sec><st>Conclusion</st>
<p>RFA of IPV may be a promising procedure, but patient and incompetent perforator vein selection is important and further standardization of the procedure is required. Comparative clinical trials between RFA and other therapies are warranted.</p>
</sec>
]]></description>
<dc:creator><![CDATA[van den Bos, R R, Wentel, T, Neumann, M H A, Nijsten, T]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.008090</dc:identifier>
<dc:title><![CDATA[Treatment of incompetent perforating veins using the radiofrequency ablation stylet: a pilot study]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>212</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>208</prism:startingPage>
<prism:section>Original articles</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/213?rss=1">
<title><![CDATA[Pyoderma gangrenosum; a rare complication of sclerotherapy]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/213?rss=1</link>
<description><![CDATA[
<p>Pyoderma gangrenosum may develop at sites of minor trauma and in surgical wounds. In susceptible people, even minimal trauma or irritation to the skin can result in the production of pyoderma gangrenosum lesions. This paper reports an unusual case of pyoderma gangrenosum following ultrasound-guided sclerotherapy.</p>
]]></description>
<dc:creator><![CDATA[De Felice, E., Allen, B.]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.009028</dc:identifier>
<dc:title><![CDATA[Pyoderma gangrenosum; a rare complication of sclerotherapy]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>214</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>213</prism:startingPage>
<prism:section>Short report</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/215?rss=1">
<title><![CDATA[Tenth Meeting of the European Venous Forum: Copenhagen, Denmark, 5-7 June 2009]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/215?rss=1</link>
<description><![CDATA[]]></description>
<dc:creator><![CDATA[Sommer, A, Shadid, N, Smith, P C., Kakkos, S, Lampropoulos, G, Papadoulas, S, Ntouvas, I, Tsolakis, I, Kalodiki, E, Adiguzel, C, Iqbal, O, Geroulakos, G., Vandendriessche, M, Lebard, C, Daniel, C, Zuccarelli, F, Uhl, J-F, Chahim, M, Casals, F J, Bernaudo, D, Lozano, N, the UFMATE Team, Sokolov, A L, Lyadov, K V, Loutsenko, M M, Lavrenko, S V, Metcalfe, M J, Gatenby, P A, Reise, J A, Franklin, J J, Chang, R, Lozier, J, Kam, A W, Chen, E A, Shawker, T H, Wesley, R A, Mao, E A, Wyrick, D A, Horne, M K, Kurtoglu, M, Ekim, H, Saba, D, Koksoy, C, Akcaly, Y, Gormuth, N, Karabay, O, Filizcan, U, Aslym, E, Dolgun, A, Mihmanly, M, Ceviz, M, Alayunt, E A, Eren, N, Broholm, R, Baekgaard, N, Just, S, Jorgensen, M, Jensen, L P, Kodati, S, Patel, V M, Makanjuola, J, Mobasheri, M, Hussain, T, McLafferty, R B, Moore, C, Hood, D, Hodgson, K J, Gasparis, A P, Labropoulos, N, Lebda, P, Spentzouris, G, Leon, L, Borge, M, Tassiopoulos, A K, Pappas, P J, Milic, D J, Zivic, S S, Bogdanovic, D C, Neglen, P, Darcey, R, Raju, S, Partsch, H, Partsch, B, Wust, M, Bahl, V, Hu, H M, Henke, P, Wakefield, T W, Campbell, D A, Caprini, J A, Cornu-Thenard, A, Uhl, J F, Carpentier, P H, Pittaluga, P, Chastanet, S, Locret, T, Gianesini, S, Menegatti, E, Tacconi, G, Palazzo, A, Liboni, A, Zamboni, P, Saarinen, J, Hjerppe, A, Venermo, M, Huhtala, H S, Vaalasti, A, Casoni, P, Nelzen, O, Mosti, G, Mattaliano, V, Partsch, H, Pichot, O, Alm, J, Vuylsteke, M, Van Dorpe, J, Roelens, J, De Bo, T., Mordon, S, Rasmussen, L H, Lawaetz, M, Bjoern, L, Lawaetz, B, Blemings, A, Eklof, B, Gale, S S, Lee, J N, Walsh, M E, Wojnarowski, D L, Comerota, A J, Ricci, S, Moro, L, Antonelli-Incalzi, R, Shepherd, A C, Gohel, M S, Lim, C S, Hamish, M, Davies, A H, Shepherd, A C, Gohel, M S, Lim, C S, Hamish, M, Davies, A H, van Rij, A M, Philips, L V, Bateman, E, Madut, D, Kleffmann, T, Jones, G, Rao, S, Konig, G, Leers, S A, Cho, J S, Marone, L K, Rhee, R Y, Makaroun, M S, Chaer, R A, Thors, A, Muck, P, Gohel, M, Lim, C S]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.09a003</dc:identifier>
<dc:title><![CDATA[Tenth Meeting of the European Venous Forum: Copenhagen, Denmark, 5-7 June 2009]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>236</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>215</prism:startingPage>
<prism:section>European Venous Forum Abstracts</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/237?rss=1">
<title><![CDATA[Lymphoscintigraphy in paediatric patients]]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/237?rss=1</link>
<description><![CDATA[]]></description>
<dc:creator><![CDATA[Bellini, C, Bonioli, E, Boccardo, F]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.009012</dc:identifier>
<dc:title><![CDATA[Lymphoscintigraphy in paediatric patients]]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>237</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>237</prism:startingPage>
<prism:section>Letter to the Editors</prism:section>
</item>

<item rdf:about="http://phleb.rsmjournals.com/cgi/content/short/24/5/238?rss=1">
<title><![CDATA[Reply to letter 'Lymphoscintigraphy in paediatric patients']]></title>
<link>http://phleb.rsmjournals.com/cgi/content/short/24/5/238?rss=1</link>
<description><![CDATA[]]></description>
<dc:creator><![CDATA[Damstra, R J, Mortimer, P S]]></dc:creator>
<dc:date>2009-09-18</dc:date>
<dc:identifier>info:doi/10.1258/phleb.2009.09r002</dc:identifier>
<dc:title><![CDATA[Reply to letter 'Lymphoscintigraphy in paediatric patients']]></dc:title>
<dc:publisher>American College of Phlebology</dc:publisher>
<prism:number>5</prism:number>
<prism:volume>24</prism:volume>
<prism:endingPage>238</prism:endingPage>
<prism:publicationDate>2009-10-01</prism:publicationDate>
<prism:startingPage>238</prism:startingPage>
<prism:section>Letter to the Editors</prism:section>
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